Astrocytic EphA4 Signaling Is Important for the Elimination of Excitatory Synapses in Alzheimer’s Disease

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Scientists showed that astrocyte-specific deletion of erythropoietin-producing hepatocellular A4 (EphA4) restored excitatory synapse loss in Alzheimer’s disease transgenic mouse models.
[Proceedings of The National Academy of Sciences of The United States of America]
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