Exosomal ETV4 Derived From M2 Macrophages Induces Growth, Glycolysis and Stemness in Hepatocellular Carcinoma by UpRegulating SULT2B1 Expression

ETV4 derived from M2 macrophage exosomes promoted hepatocellular carcinoma (HCC) cell proliferation, glycolysis and stemness in vitro, and enhanced HCC growth in nude mice.